| 1. | These ligands flowed into the space representing the synaptic cleft and bound to AChRs.
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| 2. | Pyridostigmine inhibits acetylcholinesterase in the synaptic cleft, thus slowing down the hydrolysis of acetylcholine.
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| 3. | Methylphenidate increases concentrations of dopamine and norepinephrine in the synaptic cleft, promoting increased neurotransmission.
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| 4. | These receptors are used to pass on an action potential across the synaptic cleft between neurons.
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| 5. | The localization of pikachurin to synaptic cleft in the photoreceptor ribbon synapse was determined using fluorescent antibodies.
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| 6. | Pyridostigmine decreases the degradation of acetylcholine after release into the synaptic cleft, and thereby improves muscle contraction.
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| 7. | After release into the synaptic cleft, monoamine neurotransmitter action is ended by reuptake into the presynaptic terminal.
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| 8. | The encoded protein transports the neurotransmitter glutamate into synaptic vesicles before it is released into the synaptic cleft.
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| 9. | The dopamine transporter can no longer perform its reuptake function, and thus dopamine accumulates in the synaptic cleft.
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| 10. | SERT is a type of monoamine transporter protein that transports serotonin from the synaptic cleft to the presynaptic neuron.
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